Info o účastníkovi: Current appointment:
Senior Researcher at the Department of General and Molecular Pathophysiology,
Bogomoletz Institute of Physiology,
Ukrainian National Academy of Sciences
Kiev, Ukraine.
Jun 2013 – present.
Previous appointments:
PhD Student Department of Experimental Cardiology
A.A. Bogomoletz Institute of Physiology
Ukrainian National Academy of Sciences
Kiev, Ukraine.
Oct 2004 – Mar 2008
Postdoctoral position at the Department of Biochemistry, Biophysic and Biotechnology,
Jagiellonian University, Krakow, Poland
Dec 2009 – Nov 2010
Postdoctoral position at the Institute of Heart Research, Slovak Academy of Sciences, Bratislava, Slovak Republic
Sep 2012 – June 2013.
Field of scientific interests:
Intracellular proteolysis, proteasomal proteolysis, programmed cell death, cardio-vascular research, immunology.
Hobbies: Volleyball, Literature.
Účastníkova publikačná činnosť:
Zobraziť
1. Dosenko VE, Nagibin VS, Tumanovskaya LV, Zagoriy VY, Moibenko AA, Vaage J.
Proteasome inhibitors eliminate protective effect of postconditioning in cultured neonatal
cardiomyocytes // Fiziol Zh. 2006;52(3):15-24.
2. Nagibin VS, Dosenko VE, Tumanovska LV, Moibenko AA. Protective effect of autophagy in
anoxia-reoxygenation of isolated cardiomyocyte? // Autophagy. 2006 Oct-Dec;2(4):305-6.
3. Dosenko VE, Zagorii VIu, Nagibin VS, Moibenko AA. Effect of N-3-methyladenine on
peptidase and ribonuclease activity of proteasome. // Ukr Biokhim Zh. 2005 Sep-Oct;77(5):52-6.
4. Dosenko VE, Nagibin VS, Tumanovskaia LV, Zagorii VIu, Moibenko AA. The influence of
quercetin on the activity of purified 20S, 26S proteasome and proteasomal activity in isolated
cardiomyocytes. // Biomed Khim. 2006 Mar-Apr;52(2):138-45.
5.Nagibin VS, Dosenko VE, Tumanovskaya LV, Zagoriy VY, Moibenko AA, Vaage J
Proteasomal proteolysis in anoxia-reoxygenation, preconditioning and postconditioning of
isolated cardiomyocytes. // Pathophysiology. 2006 May;13(2):119-25.
6. Surovaya OV, Dosenko VE, Nagibin VS, Tumanovskaya LV, Moibenko AA. Effects of late
postconditioning on gene expression and cell death in neonatal rat cardiomyocyte cultures. //
Pathophysiology. 2009 Jun;16(1):47-52.
7. Lisovyy OO, Dosenko VE, Nagibin VS, Tumanovska LV, Korol MO, Surova OV, Moibenko
OO.Cardioprotective effect of 5-lipoxygenase gene (ALOX5) silencing in ischemia-
reperfusion. // Acta Biochim Pol. 2009;56(4):687-94.
8. Pashevin DA, Tumanovska LV, Dosenko VE, Nagibin VS, Gurianova VL, Moibenko AA.
Antiatherogenic effect of quercetin is mediated by proteasome inhibition in the aorta and
circulating leukocytes. // Pharmacol Rep. 2011;63(4):1009-18.
9. N Tribulova, V Nagibin, T Benova, C Viczenczova, J Radosinska, V Knezl, I Dovinova,
M Barancik. Age-related deterioration of cardiac cell-to-cell coupling likely contributes to
development of atrial fibrillation in old guinea pig. // Cardiovascular Research, 2014;
103(suppl 1):S6.
10. Drevytska T., Nagibin V., Gurianova V., Moybenko O, Dosenko V. Silencing of TERT
decreases levels of miR-1, miR-21, miR-29a and miR-208a in cardiomyocytes. // Cell Biochem
Funct. 2014;32(7):565-70.
11. Nagibin VS, Kyrychenko VO, Tumanovska LV, Pashevin DO, Gurianova VL, Moibenko AA,
Dosenko VE, Klionsky DJ. Knockdown of PSMB7 induces autophagy in cardiomyocyte
cultures: possible role in endoplasmic reticulum stress. // Pathobiology. 2014; 81(1):8-14.
12. Pashevin D., Nagibin V., Tumanovska L., Dosenko V. Proteasome Inhibition Diminishes
Neutrophil Extracellular Traps Formation and Prevents Cardiomyocytes Death in Coculture
with Activated Neutrophils During Anoxia-Reoxygenation. // Pathobiology. 2015; 82(6):290-8.
13. Shysh AM, Nagibin VS, Kaplinskii SP, Dosenko VE. N-3 long chain polyunsaturated fatty
acids increase the expression of PPARγ-target genes and resistance of isolated heart and
cultured cardiomyocytes to ischemic injury. // Pharmacol Rep. 2016; 68(6):1133-1139.
14. Nagibin V, Egan Benova T, Viczenczova C, Szeiffova Bacova B, Dovinova I, Barancik M,
Tribulova N. Ageing related down-regulation of myocardial connexin-43 and up-regulation of
MMP-2 may predict propensity to atrial fibrillation in experimental animals. // Physiol Res.
2016; 65 Suppl 1:S91-S100.
Typ účastníka: Pasívny
